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SEFAYGSFVRTVSLPV details
Primary information
ID antitb_1222,
Name23679078
N-Terminal modificationPeptide
C-Terminal ModificationSEFAYGSFVRTVSLPV
Chemical ModificationConjugated with INH
Linear/CyclicFree
LengthConjugated with INH at N-terminal
ChiralityLinear
Nature16
SourceL
SpeciesProtein Derived
Strain91−106 region of the heat shock protein (Hsp16.3)
Inhibition ConcentrationNone
In Vitro/ In vivoNone
Sequence2013
CytotoxicityNA
In vivo ModelNA
Lethal DoseNA
Immune ResponceNA
Mechanism of ActionNA
TargetNA
Combination TherapyInteract with Phospholipid Langmuir Monolayers and enhnaces the cell permating abiloity of INH
Other activitiesPhospholipid Langmuir Monolayers
PMIDIsoniazide (INH)
Year of PublicationNone
Tertiary Structure
(Technique)
Not Predicted),
Primary information
ID antitb_1649,
Name19319854
N-Terminal modificationTuftsin peptide conjugate compound
C-Terminal ModificationSEFAYGSFVRTVSLPV
Chemical ModificationINH is conjugated
Linear/CyclicFree
LengthNone
ChiralityCyclic
Nature16
SourceL
OriginCationic
SpeciesNatural
StrainDerived from 16-kDa protein of M. tuberculosis and tuftsin-derived peptides
Inhibition ConcentrationMycobacterium Tuberculosis
In Vitro/ In vivoMycobacterium tuberculosis H37Rv ATCC 27294
Cell LineMIC = 2.54 μg/ml
Inhibition Concentrationin vitro
Sequence2009
CytotoxicityNA
In vivo ModelNA
Lethal DoseNA
Immune ResponceNA
Mechanism of ActionNA
TargetNA
Combination TherapyCell wall biosynthesis inhibition
Other activitiesNA
PMIDPeptide + INH(0.24 ug/ml)
Year of PublicationNA
Tertiary Structure
(Technique)
Not Predicted),
Primary information
ID antitb_1650,
Name19319854
N-Terminal modificationTuftsin peptide conjugate compound
C-Terminal ModificationSEFAYGSFVRTVSLPV
Chemical ModificationINH is conjugated
Linear/CyclicFree
LengthNone
ChiralityCyclic
Nature16
SourceL
OriginCationic
SpeciesNatural
StrainDerived from 16-kDa protein of M. tuberculosis and tuftsin-derived peptides
Inhibition ConcentrationMycobacterium Tuberculosis
In Vitro/ In vivoMycobacterium tuberculosis H37Rv ATCC 27294
Cell LineMIC = 2.26μg/ml
Inhibition Concentrationin vitro
Sequence2009
CytotoxicityNA
In vivo ModelNA
Lethal DoseNA
Immune ResponceNA
Mechanism of ActionNA
TargetNA
Combination TherapyCell wall biosynthesis inhibition
Other activitiesNA
PMIDPeptide + INH(0.24 ug/ml)
Year of PublicationNA
Tertiary Structure
(Technique)
Not Predicted),