| DB ID | MyCo_1545 |
| Title | Alternaria alternata acts on human Monocyte-derived Dendritic cells to mediate Th2/Th17 polarisation |
| Year | 2017 |
| PMID | 27823917 |
| Fungal Diseases involved | Alternaria alternata infection |
| Associated Medical Condition | Asthma |
| Genus | Alternaria |
| Species | alternata |
| Organism | Alternaria alternata |
| Ethical Statement | None |
| Site of Infection | None |
| Opportunistic invasive | None |
| Sample type | None |
| Sample source | None |
| Host Group | Human |
| Host Common name | Human |
| Host Scientific name | Homo sapiens |
| Biomarker Name | CD14 |
| Biomarker Full Name | CD14 |
| Biomarker Type | Diagnostic |
| Biomolecule | Protein |
| Geographical Location | Iran |
| Cohort | None |
| Cohort No. | None |
| Age Group | None |
| P Value | None |
| Sensitivity | None |
| Specificity | None |
| Positive Predictive Value | None |
| MIC | None |
| Fold Change | None |
| Pathway | None |
| Disease Introduction Mechanism | Asthma exacerbation is a major agent of disease for the patients involved with moderate asthma. Sensitisation and exposure to the fungal allergen Alternaria alternata (A. alternata) are a risk factor for the start of severe asthma symptoms, including threatening and mortal responses. The exclusive relation between A. alternata and exacerba- tion of asthma is clear; but the aetiology of the exclusive pathogenesis of A. alternata has not been precisely under-stood. |
| Technique | ELISA |
| Analysis Method | ELISA Based |
| ELISA kits | None |
| Assay Data | None |
| Validation Techniques used | ELISA |
| Up Regulation Down Regulation | Decrease |
| Sequence Data | None |
| External Link | None |
